Dr. Avnish Jolly,7 June:Bernie Zilbergeld, Sexual psychologist and popular author have shown that most men with potency problems believe in these advertised myths and also pointed out that many of these men engage in which the man gets stuck in observing and assessing the sexual situation as if he’s an outsider, rating the woman’s expectations and what he can deliver. And then there’s stress. But it’s not necessarily the type of stress typically portrayed by the media. Turns out the problem are rarely the work stress itself, but rather it’s transference of work performance standards to your sex life. Approaching your sex life with that same attitude; feeling pressured to perform to certain standards, can turn the act of love into another grueling task that must be completed, and completed well.
Research into the mechanics of love life is not done for some altruistic purpose. Scientists don’t necessarily care about figuring this out in order to help you understand yourself better and improve your life. Research is done to create medications that act on nervous system to stimulate desire, which is a pretense to love itself. Instead of dealing with emotional and psychological issues that are blocking from connecting with and feeling desire for partner, may soon be able to take a pill that removes the very act of needing to feel anything. One such experimental compound already under development is a peptide called bremelanotide. It works by blocking receptors in brain that are involved in regulating basic drives such as eating and sex. In human studies bremelanotide has triggered spontaneous erections in men, and boosted sexual arousal and desire in women.
As convenient as these drugs may appear, impotence drugs contribute to infertility, heart problems and can even blind you. And taking synthetic hormones in hopes of putting the spring back into step causes all sorts of terrible health problems too.
One can boost sex drive with lee intake of sugar according to a study published last year in the Journal of Clinical Investigation, high levels of sugar in bloodstream can turn off the gene that controls sex hormones. Simple glucose and fructose are metabolized in liver, with the excess stored as fat lipids. Excess fat synthesis in turn deactivates Sex Hormone Binding Globulin (SHBG) gene, maintains levels of SHBG protein to drop dramatically, and it is this SHBG protein that controls testosterone and estrogen levels.
The neurotransmitter that triggers the sexual message, in both men and women, is acetylcholine (ACH). With too little ACH, sexual activity goes down. One way to safely and effectively enhance ACH levels in your body is to take choline supplements (1,000-3,000 mg) and vitamin B5 (500-1,500 mg).
• Studies have shown active men who engage in regular physical activity lowered their risk of experiencing sexual dysfunction. So start an exercise program guys! Remember, when using exercise as a drug, it’s important to set a goal of 60 to 90 minutes per day, every day. Obviously, depending on your current condition, you may need to work slowly up to this level.
• Optimize your diet based on your body’s unique nutritional type. We each have a unique nutritional type with varying demands for the ratios of macronutrients (fats, proteins and carbohydrates) to function optimally. This is why you may respond well to a low-carb diet while your friend on the same exact diet might not.
• Because sexual dysfunction can worsen due to stress and anxiety, take control of your emotions by learning the Emotional Freedom Technique (EFT). EFT is a psychological acupressure technique that can help you effectively address your stress-related thoughts and leave you feeling calmer and more able to face your challenges, whatever they may be.
Scientific American articles linked below explore the body of research on this compelling and universal subject.
• Scientific American May 2008
http://www.sciam.com/article.cfm?id=the-orgasmic-mind&sc=rss
• Journal of Personality and Social Psychology December 2007; 93(6):1108-21
http://www.ncbi.nlm.nih.gov/pubmed/18072857?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum